It is frequently used as a salt, such as sodium oxybate (NaGHB, potassium oxybate, or Xyrem) or sodium oxybutyrate (KGHB, sodium oxybate).GHB is also created during fermentation in small amounts and can be found in various beers, wines, meat, and slight citrus fruits.
GHB is currently only used to treat narcolepsy and, less frequently, alcoholism, while there is no evidence to support this use for alcoholism from randomized controlled trials. Fibromyalgia is occasionally treated with it off-label. The active component of the prescription drug sodium oxybate (Xyrem) is buying GHB. The U.S. Food and Drug Administration has given sodium oxybate the go-ahead to treat excessive daytime sleepiness (EDS) and cataplexy associated with narcolepsy.
GHB is an intoxicant that slows down the central nervous system. The street names are numerous. Anecdotally, its effects have been compared to those of MDMA and ethanol (alcohol), including euphoria, disinhibition, increased libido, and empathogenic states. An analysis comparing the risks of ethanol and GHB came to the same conclusion. GHB may cause nausea, dizziness, sleepiness, agitation, disturbed vision, slow breathing, forgetfulness, unconsciousness, and even death at higher doses. Polydrug toxicity is one probable factor in GHB consumption-related fatalities. Alcohol and benzodiazepines, which all bind to gamma-aminobutyric acid (or “GABA”) receptor sites, can have an additive impact (potentiation) when used together with other CNS depressants.GHB’s effects can last anywhere between 1.5 and 4 hours, or even longer if high doses were used. When GHB is used with alcohol, it can result in respiratory arrest, vomiting, and unwakable slumber, all of which can have a fatal end.